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However, since the nodular melanomas in the tissue microarray series had greater mean thickness than superficial spreading melanomas, we studied securin expression in a separate series of 29 pairs of nodular and superficial spreading melanomas, matched for all histological prognostic markers including tumour thickness (Table 2). Paired t tests revealed significant (P=0.018) differences in securin expression between nodular and superficial spreading melanomas (mean number of immunoreactive cells in five HPF: nodular melanoma=48.1; superficial spreading melanoma=29.2), whereas there was no correlation between securin expression and tumour thickness.Table 2 Expression of securin and p53 in 29 pairs of nodular (NM) and superficial spreading melanomas (SSM), matched for thickness (column 3), ulceration (column 4), mitotic index (column 5; 1=2; 2=between 1 and 6/mm2; 3=>6/mm2), tumour infiltrating lymphocytes (TILS, column 6; 0=absent; 1=nonbrisk; 2=brisk) and regression (column 7).6//mm2), tumour infiltrating lymphocytes (TILS, column 6; 0=absent; 1=nonbrisk; 2=brisk) and regression (column 7) Unfortunately we are unable to provide accessible alternative text for this. In addition, no correlation was found between the number of CD31 immunoreactive microvessels and the number of securin expressing cells.hPTTG1 and Beta Catenin Expression in MelanomaZhou et al22 detected cytoplasmic accumulation of beta catenin in oesophageal carcinomas that overexpressed PTTG1 and suggested that overexpression of PTTG1 in these tumours was likely due to the activation of beta catenin/WNT signalling.
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